Wired The house mouse.
It’s the thing we call the cat.
It is, for better or worse, the symbol of catkind.
The house mouse has been around since at least the Middle Ages.
But it didn’t really make the news until the year 2000, when a group of physicists led by physicist Stephen Hawking became interested in the cat and decided to try and breed a domestic mouse, dubbed “the house mouse”.
They didn’t succeed, but in 2005, the team reported in the journal Nature that the mouse had a mutation that led to it being more like a house mouse than a cat.
The mice didn’t get much of a chance to adapt to their new home.
The scientists, led by Richard Thorne, a University of Queensland researcher, decided to start looking for an alternative.
So they set up a small breeding lab and bred mice using artificial insemination.
Their experiment was successful, and the mouse became the first species of house mouse to be successfully bred.
That was in 2005.
In 2007, Thorne and his colleagues took another look at the mouse’s genetic heritage.
This time they got more genetic diversity from the mouse, and discovered that its genes were more similar to those of other house mice than they were to those from the domestic cat.
They were even more surprised to find that the genetic variation in the mice was more common than that in the house mice.
They also discovered that the mice had a higher incidence of diseases like asthma and emphysema, as well as having a higher mortality rate.
But Thorne was not satisfied.
The genetic data from the mice, as far as he was concerned, suggested that the house-mouse mutation was responsible for a lot of the genetic differences between the two species.
It was as if they were all the same species.
So he decided to do something more ambitious.
He and his collaborators tried to find a gene called MC1R, or mitochondrial complex I, that was the genetic basis for house mice’ genetic diversity.
The gene had been linked to immunity and cancer in mice, and it was thought to have played a role in the development of human immune system.
The team then used genetic analysis of the MC1Rs of mouse and house mice to sequence their genomes.
They found that MC1r was significantly different between the species.
And, crucially, it was much higher in the mouse.
Thorne told me that this was the first time he’d seen the DNA sequence of a gene that could be linked to a disease.
He was stunned.
“It’s a gene we didn’t know existed until we did this analysis,” he said.
“We were quite amazed.”
A new species of mouse The team didn’t think they had found the missing piece to the puzzle, but they were convinced that the MCI genes were indeed a factor in the human immune systems of house mice and mice.
So Thorne started working with the other team of scientists, the Institute of Genomic Medicine in Zurich, to understand exactly how the MCIs were expressed in the cells of the mouse and mouse’s immune system, which could tell them more about how the mouse or the house could survive in different environments.
The first thing they wanted to know was whether the genes of the mice were more prevalent in the immune system of the house or the mouse itself.
To do this, they analysed the DNA of a mouse that had undergone in vitro fertilisation.
They took samples of the DNA from the immune cells of both mice, then sequenced them and used a technique called polymerase chain reaction (PCR) to identify the MCAs that were in each sample.
By analysing the PCR results, the scientists were able to determine that the genes were in different parts of the genome of the two animals.
They then looked at the genomes of the animals to see which MCAs were present in each of the animal’s cells.
By this, the researchers were able, in essence, to determine which MCA gene the mouse was expressing.
And they found that the majority of MCAs in the two mice were in the MC4R gene, which is a member of the nuclear receptor family, and which plays a key role in how the immune response works.
It also means that the mutations in the MCA2 gene were associated with immune-mediated disease.
This was something that hadn’t been found in mice before, and that’s important because MCAs are a major determinant of the development and progression of various diseases in the animal kingdom.
They showed that the mutation in MC4RA was associated with emphyscopic lung disease in mice.
This disease is caused by a defect in the ability of the immune reaction system to recognize and recognise the cells in the lungs of mice.
It can be fatal.
In addition to emphysea, emphyese and empathetic lung disease are also thought to be caused by mutations in MCAs, which are involved in the expression of genes that control the